Abbreviated AFLP, liver failure in late pregnancy, frequently of not known cause. Symptoms include sickness and sickness, stomach pain, yellowing of the skin and eyes (jaundice), frequent thirst (polydipsia), enhanced urination (polyuria), headache, and changed state of mind. Laboratory top features of AFLP feature reduced blood sugar levels (hypoglycemia), elevated liver enzymes, and lower levels of bloodstream platelets. Untreated AFLP causes full liver failure, hemorrhaging as a result of impaired blood clotting, and loss of the mother and fetus. AFLP is addressed by delivering the baby asap, usually by inducing early work. It generally subsides after distribution and does not occur in subsequent pregnancies. In some cases AFLP is involving an abnormality of fatty-acid metabolic process: a deficiency for the chemical long-chain-3-hydroxyacyl-CoA dehydrogenease (LCHAD). Mom and father have half the conventional LCHAD task, while the fetus does not have any LCHAD task. This metabolic condition into the baby's liver causes the fatty liver infection in mother.